In the beginning… There was a miracle !

With the result detailed of no pain or use of opoids needed, Dr. Williams danced and asked if he could pray. He said that he had 3 degrees, was a Hopkins doctor and these things , just don’t happen with this disease! He knew very well that this was a miracle and a gift from God.
Below is a brief summary from the paper he presented at the American Academy of Pain Medicine Conference in 2009!

Spinal cord stimulation: “neural switch” in complex regional pain syndrome type I.
Williams KA, Korto K, Cohen SP.
Source
Department of Anesthesiology and Critical Care Medicine, Johns Hopkins School of Medicine, 550North Broadway Suite 309A, Baltimore, MD 21205, USA. kwilli64@jhmi.edu
Abstract
INTRODUCTION:
Complex regional pain syndrome type I (CRPS I) is a neuropathic pain disorder of unclear etiology. It commonly follows a trivial injury and is characterized by spontaneous pain manifesting regionally that is disproportionate to the inciting event. Associated signs and symptoms include allodynia, hyperalgesia, edema, sudomotor, vasomotor abnormalities, and trophic changes. Although multiple modalities exist to treat CRPS I, significant disability, diminution in quality of life, and reduction in overall health often accompany the syndrome.
CASE:
A case of a 57-year-old man with CRPS I who was treated with spinal cord stimulation (SCS) after failing conservative therapy is presented. One month following treatment, he experienced complete symptom resolution such that stimulation was subsequently discontinued without recurrence over the 1-year follow-up period.
CONCLUSIONS:
To date there is currently no reliably validated “cure” for CRPS. There has only been one recent report where SCS resulted in the complete eradication of the signs and symptoms associated with CRPS. This series involved adolescent girls aged 11-14 years of age, who tend to have a more benign and self-limited treatment course than that seen in adults. This raises the question as to whether a “neural switch” exists, and if so, where it is located. We postulate that the inter-neuronal connections between the central and peripheral nervous systems implicated by the current pathophysiological model is the most plausible site of this “neural switch,” and that reorganization of this interface can account for the ability of SCS to effect a complete “cure” in CRPS.